 |
 |
 |
| |
How
It Works
There is No Cure for Herpes Viral Attacks
Viruses cannot be killed because they are not alive. They clearly cannot
respire, grow or reproduce on their own. Viral attacks, like the common cold
or herpes simplex, are acquired from an infected person. The tiny virus,
100,000 times smaller than a human cell, is a short protein strand similar
to a piece of DNA. Unlike bacteria and germs, viruses are not alive, do not
consume food, and cannot be killed with antibiotics.
The Common Cold viruses pass through thin cell walls. Cells, unable
to differentiate between DNA and viral protein strands, make billions of
copies. These cells burst, infesting adjacent cells. The immune system
alerted by cell damage, alters the cell walls, making it more difficult for
the virus to enter. When you have a cold, the body’s immune system floods
the area with fluids to dilute and flush out the virus. The body reacts with
runny nose, congestion, coughing, sneezing, vomiting, diarrhea, etc.
Herpes type Viruses enter soft tissues, lying dormant in the
nerves until an attack is triggered
(See - Know Your Cold Sore Triggers).
With Herpes viruses, the body flushes out viruses in the form of blisters
and weeping lesions. However, some of the herpes viruses enter or remain in
the nerves and are inhibited, lying dormant until a new attack is initiated.
The inventor of the Cold Sore Inhibitor realized that if he could discover
and simulate these nerve signals in the early stages, he could prevent the
blisters or lesions from forming. He also noticed that the same nerve signal
disruption would significantly increased the healing process of
existing Cold Sore, Shingles, and Genital Herpes lesions/blisters.
Well... after years of research, and a 5-year clinical trial - He did it!
The invention is called the BETA signal waveform, generated by the Cold Sore
Inhibitor --- protected by 50 domestic and foreign independent pending
patents.
ALSO SEE...
Instructions on how to use the Cold Sore Inhibitor for Cold Sores, Shingles,
Genital Herpes, Warts, and Molluscum Contagiosum.
|
|
 |
 |
 |
|